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However, hepatic concentrations of AQ and DEAQ are likely higher as AQ strongly accumulates in the liver ( in vivo may be lower, exposure continues for a prolonged period as the half-life of DEAQ is 10 to 12 days (Rijken et al., 2011)

Reduction in delirium is probably explained by the reduction in opioid use among patients treated with acetaminophen (although additional mechanisms, such as reduced inflammation and antipyretic effects, are certainly possible)

Elevated transaminases and GGT are significantly associated with mortality from liver disease

From a clinical perspective, this mechanism of action might be relevant

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