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10.3390/cancers11111775 44 HuY

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Schroeter ML, Abdul-Khaliq H, Sacher J, Steiner J, Blasig IE, Mueller K

In such conditions, insufficient peroxisomal fatty acid processing could increase mitochondrial substrate pressure, whereas persistent mitochondrial dysfunction would in turn impair completion of oxidative disposal, establishing a self-reinforcing loop of NEFA accumulation, ROS production, and necroinflammation (60, 61)

Moreover, the current therapeutic approaches are severely constrained by a lack of understanding of the etiology and the correlations between the environment and behaviors

Fukumoto H, Naito Z, Asano G, Aramaki T