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63 more recently argued that recombinant MSTN proteins stimulate C2C12 proliferation, emphasizing that the source of the recombinant MSTN protein can impact the outcome of an experiment

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Given the robustness of this mitochondrial response, the multifaceted nature of PGC1 regulation in cells and a modest rescue at best by mTORC1 inhibition, we suspect that there is a degree of functional redundancy built into this metabolic plasticity system that can contribute to such an adaptive response, and complete inhibition of this system will prove difficult

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Mokhtari, V., Afsharian, P., Shahhoseini, M., Kalantar, S

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