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Fatty acid oxidation protects cancer cells from apoptosis by increasing mitochondrial membrane lipids

Lifelong voluntary aerobic exercise protocols have been shown to largely prevent age- and western diet-induced vascular endothelial dysfunction and arterial stiffening throughout the lifespan in mice, and this protective effect appears to be mediated by the mitigation of mitochondrial-derived oxidative stress and inflammation [116]

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PEBP4 deficiency aggravates LPS-induced acute lung injury and alveolar fluid clearance impairment via modulating PI3K/AKT signaling pathway

Figure 1 3.1 Vascular endothelial and alveolar epithelial injury The development of sepsis-induced ARDS can be attributed to both direct and indirect lung injury ( 3.2 Cell death With the progress of scientific research, scientists have realized that the modes of cell death include but are not limited to necrosis and apoptosis necroptosis, pyroptosis, ferroptosis, lysosomal-dependent cell death, autophagy-dependent cell death and so on (98)