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Treg is an important regulatory cell for maintaining immune tolerance and shows considerable potential in treating a variety of autoimmune diseases ( Therefore, the use of different ways to induce mouse AIH can effectively simulate the clinical symptoms and serological manifestations of different degrees and different types of human AIH, and in addition, it also provides new targets and treatment ideas for the treatment of AIH

APIs of Sirt1 at H3K18 were 1.58, 1.69, and 2.32 in three independent experiments, which means that Sirt1 prefers deacetylation compared to debutyrylation at this site

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