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To exclude the possibility that IL18R1 binds as a result of the formation of the CXCL10CXCR3 complex, we did comparative co-IP and quantification among CXCL10, CXCR3, and IL18R1

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Studies have shown that both classical and trans IL-6 signals can trigger STAT3 phosphorylation and activate the JAK/STAT3 pathway in a time-dependent manner (224)

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