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While they do increase glutathione levels to some extent, the magnitude of their effect appears to be limited primarily because they cannot overcome the decrease in GSH levels that results from the loss of synthetic capacity

In a mouse AKI model, knockdown of FTH1 aggravated RM- and cisplatin-induced renal injury, suggesting that FTH1 may play a protective role against ferroptosis in the context of AKI [160, 161]

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Is that normal

Rotin D, Staub O