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This strategy holds promise for addressing the vulnerability of conventional cancer therapy resulting from the persistence of senescent cancer cells

In triple-negative breast cancer (TNBC) cells treated with isorhamnetin (IH) and chloroquine (CQ), ROS induced Drp1-dependent mitochondrial fission and apoptosis by mediating the activation and mitochondrial translocation of CaMKII

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Copper binding is primarily mediated by the histidine and glycine residues, forming a stable coordination complex

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