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In an amyotrophic lateral sclerosis (ALS) mouse model (SOD1 G93A ), the administration of KD was able to promote ATP synthesis by bypassing the inhibited complex I in the mitochondrial respiratory chain, increasing mitochondrial energy production and membrane stabilization, leading to increased motor neuron survival and improved motor function compared to normally fed mice

10.3390/jcm9020458 80 JavierI

The increase in the late apoptotic population is suggestive of necrosis or ferroptosis, suggesting that KI-induced B cell death may be mediated by a non-apoptotic pathway