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This process relies on KLF1 (Krueppel-like factor 1), FOXO3 (forkhead box O3), and E2F2 transcription factors, chromatin condensation-regulating caspase-3, DNA methylation-regulating Tet family proteins, Asxl1, cytoskeleton-regulating Rho GTPases, and p38 MAPK signaling [51, 52]

Subsequently, the second GSH molecule can bind to the H-site and the H-site bound GSH is deprotonated into the GS form

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Furthermore, the genetic induction of nicotinamide phosphoribosyltransferase (NAMPT), a salvage pathway enzyme, in primary human and mouse monocytes cofers polarization towards inflammatory M1 macrophages and similarly NAMPT inhibition blunts this polarization (61)

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