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This leads to the initiation of mitochondrial oxidative stress, which is amplified by the activation of c-Jun N-terminal kinase (JNK), resulting in mitochondrial dysfunction and opening of mitochondrial permeability transition pores

doi: 10.1016/S0168-8278(98)80211-0

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(ii) the uptake and distribution of melanosome in keratinocytes and (iii) melanin and melanosome degradation and turnover of "pigmented" keratinocytes 5

[DOI] [PMC free article] [PubMed] [Google Scholar] [154].Yao Q, Cui J, Wang J, et al

Thus, melatonin plays a crucial role as a potent antioxidant and DNA protectant against UVR-induced oxidative damage in human skin (83)