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This restores programmed cell death in cancer cell lines including SH-SY5Y neuroblastoma and U937 histiocytic lymphoma at 1-10 nanomolar concentrations, while paradoxically stimulating healthy NIH-3T3 fibroblast growth

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The effect sizes were modest but consistent

In laboratory studies, GHK-Cu is examined for its influence on extracellularmatrix-related pathways, including collagen- and glycosaminoglycan-associated readouts in controlled experimental systems