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Leonard SS, Harris GK, Shi X (2004) Metal-induced oxidative stress and signal transduction

In settings of high leptin such as the chronic ICV leptin model, known mechanisms of glucose suppression centre on glutamatergic steroidogenic factor-1 (SF1) expressing, pro-opiomelanocortin (POMC), and agouti-related protein (AgRP) neurons in the ventromedial nucleus and arcuate nucleus of the hypothalamus, which primarily suppress hepatic glucose production and stimulate glucose uptake into BAT, muscle and the heart via peripheral neural and hormonal pathways 70

Lemieux I, Pascot A, Couillard C, Lamarche B, Tchernof A, Almeras N et al

While native IGF-1 within physiological ranges appears safe, supraphysiological stimulation of IGF-1 receptor signaling through exogenous administration of potent analogs like IGF-1 DES could theoretically promote growth of existing pre-malignant or malignant cells, accelerate tumor progression, or support metastatic spread by enhancing cell survival and proliferation while inhibiting apoptosis

Normal Labs Miss Early Damage Standard labs track only late-stage changes like falling GFR or obvious protein in the urine

doi: 10.3390/ijms23116129 133 CenaHCalderPC