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Endurance training reduced oxidative damage to skeletal muscle and mitochondrial proteins in younger participants, whereas this effect was not observed in older individuals and appeared to be primarily regulated by protein degradationspecific mechanisms

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reported two proline substitutions (2P) at the apex of the central helix and HR1 that can retain the S proteins of MERS-CoV, SARS-CoV and HKU1 in the antigenically optimal pre-fusion conformation 85 (Fig

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