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Endogenous Hydrogen Sulfide and ERK1/2-STAT3 Signaling Pathway May Participate in the Association between Homocysteine and Hypertension

When GSTM3 was upregulated, the activity of ROS was obviously reduced than that in the normal group (Figures 6C,D)

To counteract this imbalance, cancer cells adapt by upregulating GSH biosynthesis, maintaining a redox equilibrium that supports survival and proliferation (Traverso et al., 2013)

Once the vulnerable plaque is raptured, the thrombotic vessels, especially small diameter vessels such as coronary arteries, can be occluded

Additionally, cells were also observed with confocal laser microscopy (Nikon C1 Laser Scanning Confocal) after staining with DAPI