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GSTM2 interacted with ASK1 and suppressed its phosphorylation and the activation of downstream p38-JNK signalling

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Por su accin biolgica, se ha estudiado por su capacidad para: estimular la reparacin de tejidos acelerar la cicatrizacin mejorar la regeneracin muscular reducir inflamacin proteger el sistema digestivo Su mecanismo lo convierte en uno de los pptidos ms relevantes para recuperacin fsica y salud articular

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