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doi:10.1161/CIRCOUTCOMES.114.001177

Clinical ASD arises from diverse etiologies including de novo mutations (e.g., SHANK3, CHD8), copy number variations, and polygenic risk factors [44]

However, considering that the majority of butyrate is metabolized as energy substrate by the colonic epithelium (12), the dosages used for treatment may be quite different between in vivo and in vitro models (4)

As a result, professional laboratories frequently implement controlled handling procedures designed to preserve formulation quality and long-term stability