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Consistently, NIK-deficient blood monocytes display inflammatory phenotype in response to bacterial lipopolysaccharide (LPS) stimulation, and NIK-deficient mice display increased pro-inflammatory myeloid cells in the blood, bone marrow and adipose tissue, although it is unclear whether this phenotype is due to macrophage-intrinsic function of NIK [26]

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J Physiol Biochem

H., Hong, J

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