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Biomed Pharmacother 105:491497

Its underlying mechanisms are multifaceted, encompassing genetic variations, environmental influences, cerebral artery constriction, abnormalities in monoamine neurotransmitters, oxidative stress, mitochondrial disturbances, the involvement of calcitonin gene-related peptide (CGRP), neurogenic inflammation, and broader neuroinflammation (6)

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Data were analyzed using the 2 Ct method with TATA box binding protein (TBP) as a housekeeping gene and expressed as fold change relative to the controls

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