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13ln for B16F10 model)

doi: 10.1186/s12935-023-02848-3

ROS directly oxidize the glutaminase protein, altering its structure and function, and modulating metabolic pathways, including elevated mitochondrial ROS production, which further affects glutaminase activity

Disorders in purine metabolism, such as a deficiency in hypoxanthine phosphoribosyl-transferase and overactivity of phosphoribosyl pyrophosphate synthetase, lead to elevated UA levels [20]

Many of the experiments reviewed here rely on pharmacologic G6PD inhibition or G6PD siRNA knockdown

A similar result was found in mice challenged with B16-F10 cells (Supplementary Fig