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Semax is a synthetic heptapeptide derived from an adrenocorticotropic hormone fragment and researched for its effects on neuronal network activity, neurotrophins (e.g., BDNF), inflammation signaling, and oxidative stress pathways

In landmark papers published over the past 25 years, Brunger and various co-authors discovered the molecular details of how synaptotagmin and other proteins cooperate to orchestrate neurotransmission

HK2-targeting peptides trigger mitochondrial Ca2 + overload, leading to Ca2 + -dependent calpain activation, mitochondrial depolarization and cell death, and can also cause massive death of chronic lymphocytic leukemia B cells (227)

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Hematoma changes during clot resolution after experimental intracerebral hemorrhage