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doi: 10.1074/jbc.RA119.008725 36 GokulanathanA.MoH

This is largely due to a lack of isoform specific inhibitors and activators and the need to target specific cells to avoid unintended effects

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It was shown that STAT5-deficient NK are able to produce increased levels of VEGF-A, suggesting that STAT5 is a repressor of NK-induced angiogenesis [47]