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But the IGF-1 LR3 reddit reviews community makes clear that the marketed promise (localized muscle growth without systemic effects) doesn't match the lived pharmacokinetic reality (full-body receptor activation with dose-limiting side effects)

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Researchers and clinicians using it do so based on extrapolation from animal data and accumulating clinical experience rather than proven efficacy in humans

We also conclude that the IMS does not contain its own GSSGreducing machinery

Moreover, according to the glutamate-glutamine-GABA cycle, astrocytes release glutamine to neurons and inhibitory neurons convert it to GABA

Both reasons are valid but the approach, expectations, and oversight should differ accordingly