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GHK-Cu is believed to be involved in multiple mechanisms of regeneration and protection in different tissues around the body

Investigating in vitro and in vivo models of intervertebral disc degeneration revealed the relationship between oxidative stress and cuproptosis, in which oxidative stress increased copper influx by enhancing the expression of CTR1 and ATP7A and subsequently promoted cuproptosis in the presence of copper by upregulating FDX1 and TCA-related proteins [58]

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Thymosin 4 activates integrin-linked kinase and promotes cardiac cell migration, survival and cardiac repair