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Antioxidative enzymes like GPx and SOD, however, tend to be dependent on the state of CD as well as the serum level

To characterize DIC in mice, we induced DIC in mice as described previously with some modification (24): administration of DOX (6 mg/kg on days 0, 2, and 4)

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This molecular explanation of ferroptotic cell death sets the stage for ferroptosis therapeutic strategies that not only act on lipid peroxidation, but that additionally target the executors of ferroptotic cell death: truncated PLs and LDEs

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There is a particular need for human studies focused on the following: (i) validation of airway and systemic GSH/GSSG redox balance as a biomarker of asthma susceptibility, (ii) the natural history of GSH/GSSG redox balance in asthma across the lifespan, (iii) the specific role of GSH/GSSG redox balance in clinical manifestations of the disorder, including asthma severity and the response to secondary insults, (iv) the utility of GSH/GSSG redox balance as a predictor and indictor of asthma treatment responsiveness, and (v) therapeutic approaches to restore the GSH/GSSG redox balance in asthma and modify the course of the disorder