The tripeptide structure of GSH suggest its potential role as a neuroactive molecule
In fact, it is significantly more toxic than alcohol itself
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10.1007/BF02211841 [DOI] [PubMed] [Google Scholar] 6.Loke YJ, Hannan AJ, Craig JM
Neuronal NO synthase (nNOS) and endothelial NO synthase (eNOS) are constitutive NO synthases, and responsible for the continuous basal release of NO
Research has shown that superoxide, a reactive oxygen species, optimizes force production in skeletal muscle ( Present perspectives Although recent publications from authors such as First, NO may affect contractile function via its involvement at the neuromuscular junction (Wildemann and Bicker, 1999), enhances neurotransmitter release ( It is well-established that NO regulates intracellular calcium via ryanodine receptor activation and sarco/endoplasmic reticulum calcium ATPase pump inhibition ( NO and mitochondria Role of NO in mitochondrial biogenesis Although the underlying mechanisms are not fully understood, several studies indicate an interaction between NO and a mitochondrial biogenesis regulator peroxisome proliferator-activated receptor gamma coactivator-1 alpha (PGC-1) (Figure 3)
