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In laboratory settings, ghk-cu peptide is studied for its role in copper-dependent peptide interactions, gene-expression signaling, and tissue-associated molecular pathways under controlled experimental conditions

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Results: Evidence indicates local estrogen dominance with progesterone resistance, pervasive immune dysregulation, and oxidative stress with iron-driven ferroptosis that particularly injures granulosa cells, alongside disease-relevant genetic/epigenetic regulators and reproductive-tract/gut microbiome dysbiosis

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