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Journal of Fish diseases

Chronic exposure to l-BMAA cyanotoxin induces cytoplasmic TDP-43 accumulation and glial activation, reproducing an amyotrophic lateral sclerosis-like phenotype in mice

For this purpose, the target protein (100 pmol of SERF2), 20 L of TALON magnetic beads (Clontech), and 1 g of the refolded RNA structure library were mixed in 1 mL of protein-binding buffer (10 mM Tris-HCl pH 7.5, 100 mM KCl, 10% glycerol, and 0.1 g/L BSA)

Ig domains are extremely common domains that are frequently involved in redox regulation (Wouters et al., 2010

264 Stress-induced microRNA-9/9* has been shown to post-transcriptionally suppress mt-tRNA-modification enzymes, leading to reduced U34 modification of non-mutant tRNAs and promoting mitochondrial dysfunction

Given the multifactorial etiology of its pathophysiology, which includes hormonal, metabolic, and immunological factors, MNT has proven to be a valid non-pharmacological therapeutic tool in the integrated management of this complex disease