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By lowering D-2HG levels, ivosidenib helps restore normal cellular differentiation that is often disrupted in IDH1-mutant cancers, such as acute myeloid leukemia (AML) and cholangiocarcinoma

Upon activation, the MLKL complex migrates to the plasma membrane, where its accumulation forms hotspots that open ion channels, causing cell swelling, membrane rupture, and the formation of pores