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Regulation of inflammation: Regulates excessive inflammation but does not dampen the signals for repair

This multi-target mechanism positions it as a promising dietary intervention, in contrast to high-fat diets that exacerbate gut dysfunction (142, 143)

It prevents the generation of free radicals in chemical toxin metabolism in the liver

This gradual approach helps minimize irritation and maximizes effectiveness

Src, Caveolin-1, and eNOS Another study looked at a different route that may affect nitric oxide signaling

To induce overexpression of LOXL2 or LOXL2-DM, cells were transduced with adenovirus (100 MOI for both AdLOXL2 and AdLOXL2-DM) for 24 h before initiating the activity assay