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Studies in obese rodents fed high-fat diets for extended periods show that diminished carnitine reserves in skeletal muscle are accompanied by marked perturbations in mitochondrial fuel metabolism, including reduced complete fatty acid oxidation, elevated incomplete -oxidation, and impaired substrate switching from fatty acid to pyruvate oxidation

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Rossignol and Frye have performed a systematic review and meta-analysis to investigate the prevalence of mitochondria-associated genetic abnormalities in autistic children [35]

In a recent study, MCT was administered to 20 Japanese patients with mild to moderate AD over 12 weeks, and 120 min after MCT intake, the level of KBs increased

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doi: 10.1016/j.jneuroim.2014.11.019