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Belangrijke Onderzoeksgebieden Structureel Collageensynthese Dynamiek van de extracellulaire matrix Bindweefsel-signalering Epitheliaal Matrixremodellering Weefseldynamiek Follikelsignalering Systemisch Cytokinemodulatie Cellulaire levensvatbaarheid Pathway-dynamiek Samen suggereren deze bevindingen een brede experimentele potentie voor GHK-Cu binnen structurele, epitheliale en systemische pathways

p62 participates in the inhibition of NF-B signaling and apoptosis induced by sulfasalazine in human glioma U251 cells

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The intertwined dysregulation of copper and iron metabolism further exacerbates the disease by promoting apoptosis in lipid-rich foam cells and VSMCs, which are particularly vulnerable to metal-dependent cell death [195, 196]