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Activating GLP-1, GIP, and glucagon receptors simultaneously puts more stress on the gastrointestinal system during the adaptation period

Demontis F, Patel VK, Swindell WR, Perrimon N

Nalkiran I, Turan S, Arikan S, Kahraman OT, Acar L, Yaylim I, Ergen A

doi: 10.3390/toxics13040248 124 YangBWangZWangSLiX

ER stress is a typical trait in DbCM, and a recent study demonstrated that induction of ER stress by thapsigargin treatment in non-diabetic mice impairs mitochondrial respiration, may induce mPTP opening, and increases mitochondrial oxidative stress [137]

Concomitantly, serine/threonine phosphatases and phosphotyrosine phosphatases are prone to oxidation-induced inactivation