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Overall, strategies aiming to modulate the CCA TIME must shift the balance from immunosuppressive factors in favour of cytotoxic elements to enhance the antitumour response

Core facilities at The Jackson Laboratory were supported by the US National Cancer Institute (CA034196)

Redox regulation by protein S-Glutathionylation: from molecular mechanisms to implications in health and disease

ER stress, particularly through the PERK-eIF2-ATF4 pathway which leads to increased levels of CHOP, can contribute to ferroptosis

ScientificWorldJournal 2013: 767482

In common, physical or psychological stresses cause stress and disrupt homeostasis