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Dysregulated signaling pathways such as Wnt/-catenin, TGF-, and PI3K/Akt have been shown to modulate osteoblast and osteoclast activity in OA subchondral bone

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To mechanistically determine whether 15LO1 pathway activation alters the intracellular redox balance, particularly under T2 conditions, these ex vivo findings were modeled in vitro in primary HAECs

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