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Angelman syndrome (AS) , characterized by microcephaly, seizures, motor dysfunction, and mental retardation, is a result of maternal chromosome deletions in the region 15q11-q13 associated with ubiquitin-protein ligase E3A (UBE3A) critical region [379] Angelman syndrome (AS) UBE3A encodes E6-associated protein (E6-AP), which acts as a cellular ubiquitin ligase and establishes a covalent linkage between a 76-amino acid ubiquitin molecule and its target protein to form a polyubiquitylated substrate [379]

[DOI] [PMC free article] [PubMed] [Google Scholar] 130.Certo M, Tsai CH, Pucino V, Ho PC, Mauro C

Our study found that ferroptosis is mainly involved in the early stage after ph-IOP injury

(jofem.org) Is AOD-9604 better than semaglutide or tirzepatide

Ferroptosis is a new form of non-apoptotic cell death marked by the oxidative modification of phospholipid membranes via an iron-dependent mechanism [14]

The resistance across the epithelial layer (TEER) and expression and distributions of the proteins involved in tight junctions (ZO-1, occludin, and claudin-1), receptors responsible for detecting pathogens (TLR2 and TLR4), and molecules that inhibit the TLR signaling pathway increased (A20 and IRAK-M) (186)