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L.et al (1994)

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This is potentially due to the formation of inhibitory fragments by brush-border enzymatic degradation of LA during the ex vivo recovery process

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Our own immunohistochemical analysis of 4-HNE-His levels in rat retinas after 4 months of experimental diabetes found no evidence for accumulation of this adduct (127)

The acceleration of wound healing by BPC-157 (800 ng/mL) became remarkably significant on days 12 and 16, showing an average healing of 54.38%11.37% and 77.53%10.25%, respectively