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At the molecular level, TLR signaling is critically dependent on adaptor proteins, primarily myeloid differentiation primary response 88 (MyD88) and TIR-domain-containing adaptor-inducing interferon- (TRIF), which determine the specificity, magnitude, and temporal dynamics of downstream inflammatory responses

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In diesen sensiblen Phasen knnen die schtzenden Eigenschaften des Stoffs nmlich die Wirkung von Therapien oder biologischen Prozessen beeinflussen