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Moreover, PBP surface modification enhanced targeting specificity, reduced immunogenicity, and prolonged sustained drug release in vivo , offering a promising strategy for integrated UC diagnosis and therapy

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Mitophagy is impaired in HF, causing the accumulation of damaged mitochondria, due in part to reduced PGC-1 expression, and overexpression of PGC-1 restores mitochondrial function and cardiac performance in HF models [18]

however, this effect is significantly blunted in cells deficient for CI and CIV (Fig

References Siegel RL, Miller KD, Fuchs HE, Jemal A