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Interestingly, NAD(P)H quinone dehydrogenase 1 deletion (an oxidative stress barrier) enhances the effect of rapamycin and leads to increased tuberous sclerosis complex 2 phosphorylation, indicating that autophagy may be activated to counter the increased stress and protect against AKI [63]

Plant alkaloids such as berberine carried out the inhibition of NADPH oxidase activity via reducing the mRNA expression of gp91phox in macrophages

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