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The validity of the structural models is also supported by a previous observation that mutations in F262, F266, and L269 of AAGAB disrupted its binding to the subunit in vitro using recombinant proteins ( 26 )

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It is intriguing how reactive oxygen and nitrogen species (RONS) produced by plasma interact on a molecular level in a biological environment

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SeNPs inhibits keratinocytes hyperproliferation by induction of apoptosis The homeostasis imbalance among cell differentiation, cell proliferation, and programmed cell death leads to keratinocyte hyperproliferation with shortened cell cycles [45, 46]