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Sun WX, Huang WX, Liu CH, Chen JT

Manios, Y

Abu Abed OS, Chaw C, Williams L, Elkordy AA

doi: 10.1016/j.schres.2020.05.044 195 ClarkSMNotarangeloFMLiXChenSSchwarczRTonelliLH

Polymers (Basel) 12 , 1800

This paper reviews the Met metabolism in normal conditions and its dysregulation under abnormal conditions, the possible causes of hypermethioninemia, and its connection to BA pathogenesis: Abnormal hepatic level of Met could lead to a perturbation of redox homeostasis and mitochondrial functions of hepatocytes, enhancement of viral infectivity, and dysregulation of innate and adaptative immune cells in response to infection/damage of the liver contributing to the initiation/progression of BA