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Rats treated with the drug, as compared to a control group, showed significant improvement after less than one week of treatment, which continued for the remainder of the drug application and, for an additional 12 weeks after withdrawal from the drug, indicating sustained neurological amelioration of stroke-induced cognitive deficits

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At the same time, IGF1R activates the PI3K/Akt signaling pathway, promotes Bcl-2/Bax expression and inhibits cell apoptosis (Katayama et al., 2007) (Figure 4B)

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