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Alveolar macrophages: plasticity in a tissue-specific context

Studies have shown that in the absence of GPX4, the calcium-independent phospholipase iPLA2 acts as a major ferroptosis suppressor in a GPX4-independent manner (Chen et al

The ability of ESA to enhance the kinetics of cell death when GPX4 is maximally inhibited shows that ESA and GPX4 inhibitors trigger ferroptosis by distinct mechanisms

doi: 10.1007/s00345-018-2436-y 61 FletcherS

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