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High-affinity kainate receptor subunits are necessary for ionotropic but not metabotropic signaling

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This targeted delivery paradigm not only amplifies on-target protein degradation efficacy but also mitigates the hook effect and reduces on-target/off-tumor toxicityaddressing core shortcomings of conventional PROTACs through design-driven innovation

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doi: 10.1136/annrheumdis-2018-213103 46

revealed that IL-22 and TH17 pathway could alleviate metabolic disorders in diabetes (Wang et al., 2014), the role of IL-22 and IL-23 in AS remains to be determined