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Methotrexate induces adenosine release Our laboratory originally proposed the hypothesis that the beneficial effects of methotrexate result from the intracellular accumulation of intermediates in purine biosynthesis that, by a mechanism that has not been completely worked out, leads to increased concentrations of adenosine in the extracellular space [31]

However, after using the autophagy inhibitors chloroquine and wortmannin or knocking down Bclin1 and Atg5 by siRNA to inhibit autophagy, the anticancer activity of IVM significantly decreased

PediatricAge is 0-17 years inclusive (for example, Reye's syndrome, routine child health exam)

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it could be reduced within the ER by a glutathione reductase, it could be transported to the cytosol for reduction, or it could be secreted from the cell