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Are there protective enzymatic pathways to regulate high local nitric oxide (NO) concentrations in cells under stress conditions

Different environmental stressors, including high salinity, low temperature, and drought, can activate DHAR expression, which goes on to regulate GSH pools (Urano et al., 2000

[1] That acyl modification is not incidental chemistry: it is installed by a dedicated enzyme (ghrelin O-acyltransferase) and is required for the peptide to bind and activate GHSR1a

AuNPs and their applications have been extensively studied for over five decades and have shown great promise as theranostic agents in preclinical [5, 11,12,13] and clinical studies [14,15,16,17,18]