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Hepatology 53:974982 McGill MR, Sharpe MR, Williams CD, Taha M, Curry SC, Jaeschke H (2012) The mechanism underlying acetaminophen-induced hepatotoxicity in humans and mice involves mitochondrial damage and nuclear DNA fragmentation

N6-methyladenosine demethylase FTO promotes M1 and M2 macrophage activation

uralensis , which implied that -glucosidase could involve in the starch and sucrose metabolism pathway through affecting hydrolytic activity

doi: 10.1007/s12602-022-09909-5 27 BaiX.ZhuM.HeY.WangT.TianD.ShuJ

Methods: Tamoxifen-resistant cell lines previously developed and characterized in our laboratory were metabolically profiled with nuclear magnetic resonance spectroscopy (NMR) using cryogenic probe, and the findings were correlated with the expression of genes that encode the key enzymes of the significant metabolites

At a higher dose of fluconazole 200 mg, incremental S-CIT dose adjustment by 25% did not yield the doses that satisfy the equivalent S-CIT exposure for G-EM and G-IM