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Microglia polarization from M1 to M2 in neurodegenerative diseases

Acknowledgements This research was financially supported by the POS Salute 20142020 of the Italian Ministry of Health, under Trajectory 3 Regenerative, Predictive and Personalized Medicine, Action Line 3.1, within the project Integrated Health and Genetics System for Cardiovascular Diseases (SISAGEN-CARDIO) (local project code T3-AN-18)

doi: 10.1038/nri2506 47 VegliaFSansevieroEGabrilovichDI

However, when ER stress is persistent and severe, UPR is insufficient to restore ER function, it promotes cell apoptosis by upregulating the expression of GADD153/CHOP, by activating caspase-12, and by inhibiting Bcl-2 expression

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For instance, studies have shown higher NK presence in tumors to be associated with lower pyruvate to lactate conversion, an effect hypothesized to be mediated by overexpression of FOXD1 via USP21