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Because the cells are supplied with the raw, rate-limiting materials rather than the finished product, they can auto-regulate their own glutathione synthesis

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While this may understandably retard cell division to some extent, we did not expect it to trigger substantial cellular senescence because in our experiments, IR was delivered at doses that are estimated to inflict fewer than a single DSB per cell each hour

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Furthermore, damaged mitochondria leak even more ROS into the cell, exacerbating the oxidative stress and accelerating cellular aging and dysfunction